Substance Use and Epigenetic Age Acceleration in Adults
This study examines the associations between substance use, including alcohol, tobacco, and opioids, and biological aging in a population-based sample of adults.
This study examines the associations between substance use, including alcohol, tobacco, and opioids, and biological aging in a population-based sample of adults.
This study explores the associations between substance use, including alcohol and opioids, and biological aging. Findings suggest significant links, particularly with cigarette smoking and opioid use.
A study investigates the relationship between substance use and epigenetic age acceleration, highlighting associations with alcohol, tobacco, and opioid use among adults.
A study examines the association between substance use, including alcohol and opioids, and biological aging. Findings highlight the impact of smoking and opioid use on epigenetic age acceleration.
This study explores how blood cell composition affects DNA methylation age and age acceleration, revealing distinct biological interpretations and modest impacts on health outcomes.
This study explores how blood cell composition affects DNA methylation age and its association with health outcomes, revealing distinct biological interpretations.
This study explores a deep-learning derived SASP Score as a biomarker for systemic cellular senescence burden, predicting mortality and chronic conditions in humans.
This study explores how blood cell composition affects DNA methylation age and age acceleration, revealing distinct biological interpretations and their implications for health outcomes.
This study explores a deep-learning derived SASP Score as a biomarker for systemic cellular senescence burden, predicting mortality and chronic conditions.
This study explores how blood cell composition influences DNA methylation age and age acceleration, revealing distinct biological interpretations and modest associations with health outcomes.