Blood Cell Composition Influences DNA Methylation Age and Age Acceleration
This study explores how blood cell composition affects DNA methylation age and its implications for health outcomes, revealing distinct biological interpretations.
This study explores how blood cell composition affects DNA methylation age and its implications for health outcomes, revealing distinct biological interpretations.
A study investigates the associations between substance use, including alcohol and opioids, and biological age acceleration, highlighting significant links with smoking and opioid use.
This study explores how blood cell composition affects DNA methylation age and age acceleration, revealing distinct biological interpretations and modest associations with health outcomes.
A study presents a novel DNA methylation clock that identifies immune-metabolic aging signatures associated with mortality, utilizing advanced modeling techniques.
A study investigates the relationship between substance use, including alcohol and opioids, and biological aging, revealing significant associations with epigenetic age acceleration.
This study explores how blood cell composition affects DNA methylation age and its association with health outcomes, revealing distinct biological interpretations.
This study explores a new DNA methylation clock model that predicts all-cause mortality using epigenetic data, highlighting immune-metabolic aging signatures.
This study explores the relationship between various substance uses, including alcohol and opioids, and biological aging, revealing significant associations with epigenetic age measures.
This study explores how blood cell composition impacts the interpretation of DNA methylation age and age acceleration, revealing distinct biological processes.
This study develops a DNA methylation clock that identifies immune-metabolic aging signatures associated with mortality, utilizing data from the Framingham Heart Study.